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From the international Amalgam Mailing list :
09 - 02 - 1999
Here are a couple of abstracts of studies
at UK directly contradictory
to the
one ADA is supporting:   How can they get
directly contradictory results???

Authors: Thompson CM; Markesbery WR; Ehmann
WD; Mao YX; Vance DE;
Title: Regional brain trace-element studies in
Alzheimer's disease.
Address: Department of Chemistry, University
of Kentucky, Lexington
Journal: Neurotoxicology, 9: 1, 1988 Spring,
Alzheimer's disease (AD) brain trace-element
imbalances in the
amygdala, hippocampus and nucleus basalis of
Meynert (nbM) are
found in most cases to be consistent with
those previously reported
in samples derived principally from AD
cerebral cortex (Ehmann et
al., 1986). The elevation of mercury in AD
nbM, as compared to
age-matched controls, is the largest
trace-element imbalance observed
to date in AD brain. In addition to the
general confirmation of
imbalances for Cs, Hg, N, Na, P, and Rb noted
previously in
cerebral cortex samples, imbalances for Fe, K,
Sc, and Zn were
observed in two regions and one region also
exhibited imbalances
for both Co and Se. Persistent imbalances for
the univalent cations
Na, K, Rb and Cs support arguments for a
membrane abnormality in AD.
The data presented here also provide the first
simultaneous multi-element determinations in
both control and AD nbM.

Wenstrup D; Ehmann WD; Markesbery WR; Trace
element imbalances
in isolated subcellular fractions of
Alzheimer's disease brains.
Department of Chemistry, University of
Kentucky, Lexington. Brain
Res, 533: 1, 1990 Nov 12, 125-31
Concentrations of 13 trace elements (Ag, Br,
Co, Cr, Cs, Fe,
Hg, K, Na, Rb, Sc, Se, Zn) in isolated
subcellular fractions (whole
brain, nuclei, mitochondria, microsomes) of
temporal lobe from
autopsied Alzheimer's disease (AD) patients
and normal controls
were determined utilizing instrumental neutron
activation analysis.
Comparison of AD and controls revealed
elevated Br (whole brain)
and Hg (microsomes) and diminished Rb (whole
brain, nuclear
and microsomes), Se (microsomes) and Zn
(nuclear) in AD. The
elevated Br and Hg (Mercury) and diminished Rb
are consistent with
our previous studies in AD bulk brain
specimens. Comparison of
element ratios revealed increased Hg/Se, Hg/Zn
and Zn/Se
mass ratios in AD. Se and Zn play a protective
role against
Hg toxicity and our data suggest that they are
utilized to
detoxify Hg in the AD brain. Overall our
studies suggest that
Hg could be an important toxic element in AD.
Whether Hg
deposition in AD is a primary or secondary
event remains to
be determined.

This is dated Dec 1996 - quite recent. Dr
Haleys is a biochemist
of world repute - his work is already well
known. He is a member
of the University of Kentucky's huge
multi-million dollar
long-term research project examining the
causes a Alzheimers
Disease - there are probably very few more
eminent or knowledgable
in this field. Dr Murray Vimy is also a
researcher of repute, and
a former World Health Organisation consultant.
The journal from
which the full extract is quoted (FASEB) is a
highly regarded
and main-stream scientific medical

The following quote is from The Valley
Advocate, December 5, 1996.

"A team of scientists led by Dr. Boyd Haley
recently completed a study
exposing six laboratory rats to a typical
intake of amalgam mercury
vapor, diluted to account for the size
difference between rats and
humans. To the researchers' astonsihment,
every rat developed symptoms
and brain tissue damage indistinguishable from
that of Alzheimer's
Disease patients. The reseachers then repeated
their experiment only
to find the same results.

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